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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
Fecha : |
21/02/2014 |
Actualizado : |
21/09/2016 |
Tipo de producción científica : |
Documentos |
Autor : |
DIAZ, R.; CORSI, W.; RIOS, A.; ZERBINO, M.S.; PEREA, C.; DÍAZ, M.; VERGES, R.; GERMAN, S.; ABADIE, T.; TROCHE, L. |
Afiliación : |
ROBERTO MIGUEL DIAZ ROSSELLO, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"), Uruguay; WALTER C. CORSI, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"), Uruguay; AMALIA RIOS GARCIA, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"), Uruguay; MARIA STELLA ZERBINO BARDIER, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"), Uruguay; CARLOS F. PEREA, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"); MARTHA G. DÍAZ, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"); RUBEN PIERRE VERGES DELGADO, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"), Uruguay; SILVIA ELISA GERMAN FAEDO, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"), Uruguay; TABARE E. ABADIE, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"), Uruguay; LILIAN TROCHE, CIAAB (Centro de Investigaciones Agrícolas "Alberto Boerger"), Uruguay. |
Título : |
Reseña de la producción e investigación de trigo en el Uruguay. |
Fecha de publicación : |
1982 |
Fuente / Imprenta : |
Colonia (Uruguay): CIAAB, 1982. |
Páginas : |
13 p. |
Serie : |
(CIAAB Miscelánea ; 44) |
Idioma : |
Español |
Notas : |
Informe presentado en la Reunión de Coordinadores Nacionales del Proyecto Trigo del Convenio IIC Cono Sur/BID en La Estanzuela, del 11 al 13 de Agosto de 1981. |
Thesagro : |
APLICACION DE ABONOS; CLIMA; CREDITO; DATOS DE PRODUCCION; DISTRIBUCION GEOGRAFICA; ENFERMEDADES DE LAS PLANTAS; ESCARDA; EXPORTACIONES; FINANCIAMIENTO; FITOMEJORAMIENTO; IMPORTACIONES; MERCADEO; PLAGAS DE PLANTAS; TRITICUM AESTIVUM; URUGUAY; UTILIZACION DE LA TIERRA; VARIEDADES. |
Asunto categoría : |
-- |
URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/6076/1/CIAAB-Miscelanea-44-1982.pdf
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Marc : |
LEADER 01294nam a2200445 a 4500 001 1040377 005 2016-09-21 008 1982 bl uuuu u00u1 u #d 100 1 $aDIAZ, R. 245 $aReseña de la producción e investigación de trigo en el Uruguay. 260 $aColonia (Uruguay): CIAAB$c1982 300 $a13 p. 490 $a(CIAAB Miscelánea ; 44) 500 $aInforme presentado en la Reunión de Coordinadores Nacionales del Proyecto Trigo del Convenio IIC Cono Sur/BID en La Estanzuela, del 11 al 13 de Agosto de 1981. 650 $aAPLICACION DE ABONOS 650 $aCLIMA 650 $aCREDITO 650 $aDATOS DE PRODUCCION 650 $aDISTRIBUCION GEOGRAFICA 650 $aENFERMEDADES DE LAS PLANTAS 650 $aESCARDA 650 $aEXPORTACIONES 650 $aFINANCIAMIENTO 650 $aFITOMEJORAMIENTO 650 $aIMPORTACIONES 650 $aMERCADEO 650 $aPLAGAS DE PLANTAS 650 $aTRITICUM AESTIVUM 650 $aURUGUAY 650 $aUTILIZACION DE LA TIERRA 650 $aVARIEDADES 700 1 $aCORSI, W. 700 1 $aRIOS, A. 700 1 $aZERBINO, M.S. 700 1 $aPEREA, C. 700 1 $aDÍAZ, M. 700 1 $aVERGES, R. 700 1 $aGERMAN, S. 700 1 $aABADIE, T. 700 1 $aTROCHE, L.
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INIA La Estanzuela (LE) |
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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
Fecha actual : |
29/10/2020 |
Actualizado : |
27/01/2021 |
Tipo de producción científica : |
Artículos en Revistas Indexadas Internacionales |
Circulación / Nivel : |
Internacional - -- |
Autor : |
GIANNITTI, F.; GARCÍA, J.P.; ROOD, J.I.; ADAMS, V.; ARMENDANO, J.I.; BEINGESSER, J.; UZAL, F.A. |
Afiliación : |
FEDERICO GIANNITTI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; JORGE P. GARCÍA, Universidad Nacional del Centro de la Provincia de Buenos Aires (UNCPBA), Tandil, Buenos Aires, Argentina.; JULIAN I. ROOD, Monash University, Clayton, Victoria, Australia.; VICKI ADAMS, Monash University, Clayton, Victoria, Australia; JOAQUÍN I. ARMENDANO, Universidad Nacional del Centro de la Provincia de Buenos Aires (UNCPBA), Tandil, Buenos Aires, Argentina.; JULIANN BEINGESSER, University of California at Davis, San Bernardino, CA, USA.; FRANCISCO A. UZAL, University of California at Davis, San Bernardino, CA, USA. |
Título : |
Cardiopulmonary Lesions in Sheep Produced by Experimental Acute Clostridium Perfringens Type D Enterotoxemia. |
Fecha de publicación : |
2021 |
Fuente / Imprenta : |
Veterinay Pathology, volumen 58, number 1, pag. 103-113, 2021. Doi: https://doi.org/10.1177/0300985820965554 |
DOI : |
10.1177/0300985820965554 |
Idioma : |
Inglés |
Notas : |
Article history: First Published October 15, 2020. Corresponding Author:Francisco A. Uzal, Email: fuzal@cahfs.ucdavis.edu. The author(s) disclosed receipt of the following financial support for the research, authorship, and/or publication of this article: Federico Giannitti acknowledges support from the Uruguayan ?Agencia Nacional de Investigaci ´on e Innovaci ´on? (ANII) through mobility grant MOV_CA_2018_1_150021. This work was supported by Grant R01 AI056177 from the National Institute of Allergy and Infectious Diseases (NIAID). Research at Monash University was also supported by funding provided by the Australian Research Council to the Australian Research Council Centre of Excellence in Structural and Functional Microbial Genomics (Grant CE0562063). |
Contenido : |
Abstract:Enterotoxemia caused by Clostridium perfringens type D is one of the most prevalent clostridial diseases of sheep. The lesions of
the acute form of this disease, particularly the cerebral lesions, are well characterized; however, detailed descriptions of the cardiac and pulmonary lesions are lacking. Here we describe cardiopulmonary lesions in experimental acute type D enterotoxemia in sheep and determine the role of epsilon toxin (ETX) in the development of these lesions. Four groups of 6 sheep were intraduodenally inoculated with either a wild-type C. perfringens type D strain; its etx knockout mutant, which is unable to produce ETX; the etx mutant complemented with the wild-type etx gene, which regains the ETX toxigenic ability; or sterile culture
medium as a control. All sheep were subjected to postmortem examination within 24 hours of inoculation. Lesion scores were compared between groups for pulmonary edema; hydrothorax; ascites; hydropericardium; endocardial, myocardial and epicardial hemorrhages; microscopic lesions of acute myocardial degeneration and necrosis; and myocardial, endocardial, and epicardial edema, hemorrhage, and inflammation. Only sheep inoculated with the wild-type and complemented ETX-toxigenic bacterial strains developed cardiopulmonary lesions, which were present in varying degrees of severity and proportions. These lesions were not present in sheep inoculated with the etx mutant or in the negative control. We conclude that severe acute cardiopulmonary lesions in sheep with experimental enterotoxemia are associated with the capacity of the strains to produce ETX. These changes are likely contributors to the clinical signs and even death of affected animals. MenosAbstract:Enterotoxemia caused by Clostridium perfringens type D is one of the most prevalent clostridial diseases of sheep. The lesions of
the acute form of this disease, particularly the cerebral lesions, are well characterized; however, detailed descriptions of the cardiac and pulmonary lesions are lacking. Here we describe cardiopulmonary lesions in experimental acute type D enterotoxemia in sheep and determine the role of epsilon toxin (ETX) in the development of these lesions. Four groups of 6 sheep were intraduodenally inoculated with either a wild-type C. perfringens type D strain; its etx knockout mutant, which is unable to produce ETX; the etx mutant complemented with the wild-type etx gene, which regains the ETX toxigenic ability; or sterile culture
medium as a control. All sheep were subjected to postmortem examination within 24 hours of inoculation. Lesion scores were compared between groups for pulmonary edema; hydrothorax; ascites; hydropericardium; endocardial, myocardial and epicardial hemorrhages; microscopic lesions of acute myocardial degeneration and necrosis; and myocardial, endocardial, and epicardial edema, hemorrhage, and inflammation. Only sheep inoculated with the wild-type and complemented ETX-toxigenic bacterial strains developed cardiopulmonary lesions, which were present in varying degrees of severity and proportions. These lesions were not present in sheep inoculated with the etx mutant or in the negative control. We conclude that severe acute ... Presentar Todo |
Palabras claves : |
CLOSTRIDIUM PERFRINGENS TYPE D; EPSILON TOXIN; HEART; LUNGS; PATHOLOGY; PLATAFORMA SALUD ANIMAL; SHEEP. |
Thesagro : |
ENFERMEDADES DE LOS ANIMALES; OVEJA. |
Asunto categoría : |
-- |
Marc : |
LEADER 03466naa a2200325 a 4500 001 1061439 005 2021-01-27 008 2021 bl uuuu u00u1 u #d 024 7 $a10.1177/0300985820965554$2DOI 100 1 $aGIANNITTI, F. 245 $aCardiopulmonary Lesions in Sheep Produced by Experimental Acute Clostridium Perfringens Type D Enterotoxemia.$h[electronic resource] 260 $c2021 500 $aArticle history: First Published October 15, 2020. Corresponding Author:Francisco A. Uzal, Email: fuzal@cahfs.ucdavis.edu. The author(s) disclosed receipt of the following financial support for the research, authorship, and/or publication of this article: Federico Giannitti acknowledges support from the Uruguayan ?Agencia Nacional de Investigaci ´on e Innovaci ´on? (ANII) through mobility grant MOV_CA_2018_1_150021. This work was supported by Grant R01 AI056177 from the National Institute of Allergy and Infectious Diseases (NIAID). Research at Monash University was also supported by funding provided by the Australian Research Council to the Australian Research Council Centre of Excellence in Structural and Functional Microbial Genomics (Grant CE0562063). 520 $aAbstract:Enterotoxemia caused by Clostridium perfringens type D is one of the most prevalent clostridial diseases of sheep. The lesions of the acute form of this disease, particularly the cerebral lesions, are well characterized; however, detailed descriptions of the cardiac and pulmonary lesions are lacking. Here we describe cardiopulmonary lesions in experimental acute type D enterotoxemia in sheep and determine the role of epsilon toxin (ETX) in the development of these lesions. Four groups of 6 sheep were intraduodenally inoculated with either a wild-type C. perfringens type D strain; its etx knockout mutant, which is unable to produce ETX; the etx mutant complemented with the wild-type etx gene, which regains the ETX toxigenic ability; or sterile culture medium as a control. All sheep were subjected to postmortem examination within 24 hours of inoculation. Lesion scores were compared between groups for pulmonary edema; hydrothorax; ascites; hydropericardium; endocardial, myocardial and epicardial hemorrhages; microscopic lesions of acute myocardial degeneration and necrosis; and myocardial, endocardial, and epicardial edema, hemorrhage, and inflammation. Only sheep inoculated with the wild-type and complemented ETX-toxigenic bacterial strains developed cardiopulmonary lesions, which were present in varying degrees of severity and proportions. These lesions were not present in sheep inoculated with the etx mutant or in the negative control. We conclude that severe acute cardiopulmonary lesions in sheep with experimental enterotoxemia are associated with the capacity of the strains to produce ETX. These changes are likely contributors to the clinical signs and even death of affected animals. 650 $aENFERMEDADES DE LOS ANIMALES 650 $aOVEJA 653 $aCLOSTRIDIUM PERFRINGENS TYPE D 653 $aEPSILON TOXIN 653 $aHEART 653 $aLUNGS 653 $aPATHOLOGY 653 $aPLATAFORMA SALUD ANIMAL 653 $aSHEEP 700 1 $aGARCÍA, J.P. 700 1 $aROOD, J.I. 700 1 $aADAMS, V. 700 1 $aARMENDANO, J.I. 700 1 $aBEINGESSER, J. 700 1 $aUZAL, F.A. 773 $tVeterinay Pathology, volumen 58, number 1, pag. 103-113, 2021. Doi: https://doi.org/10.1177/0300985820965554
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